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\u6e24\u5065\u516c\u53f8\u516c\u5171\u4e8b\u52a1\u90e8 \u00a0\u90ae\u7bb1:public.affairs@biogen.com<\/p>\n

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\u53c2\u8003\u6587\u732e<\/p>\n

    \n
  1. Eisai presents full results of lecanemab Phase 3 confirmatory Clarity AD study for early Alzheimer’s disease<\/li>\n<\/ol>\n

    at Clinical Trials on Alzheimer’s Disease (CTAD) conference. Available at:<\/p>\n

    https:\/\/www.eisai.co.jp\/news\/2022\/news202285.html<\/p>\n

      \n
    1. van Dyck. C, et al. Lecanemab in Early Alzheimer\u2019s Disease. The New England Journal of Medicine. DOI:<\/li>\n<\/ol>\n

      10.1056\/NEJMoa2212948. https:\/\/www.nejm.org\/doi\/full\/10.1056\/NEJMoa2212948<\/p>\n

        \n
      1. Secretar\u00eda de Salud. Enfermedad de Alzheimer, demencia m\u00e1s com\u00fan que afecta a personas adultas mayores.<\/li>\n<\/ol>\n

        Oct 2021. https:\/\/www.gob.mx\/salud\/es\/articulos\/enfermedad-de-alzheimer-demencia-mas-comun-queafecta-<\/p>\n

        a-personas-adultas-mayores?idiom=e<\/p>\n

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        1. Amin L, Harris DA. A\u03b2 receptors specifically recognize molecular features displayed by fibril ends and<\/li>\n<\/ol>\n

          neurotoxic oligomers. Nat Commun. 2021;12:3451. doi:10.1038\/s41467-021-23507-z<\/p>\n

            \n
          1. Ono K, Tsuji M. Protofibrils of Amyloid-\u03b2 are Important Targets of a Disease-Modifying Approach for<\/li>\n<\/ol>\n

            Alzheimer’s Disease. Int J Mol Sci. 2020;21(3):952. doi: 10.3390\/ijms21030952. PMID: 32023927; PMCID:PMC7037706.<\/p>\n","protected":false},"excerpt":{"rendered":"

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